風(fēng)疹-特異IgG ELISA試劑盒

風(fēng)疹-特異IgG ELISA試劑盒

廣州健侖生物科技有限公司

廣州健侖長期供應(yīng)各種ELISA試劑盒，主要代理進(jìn)口和國產(chǎn)品牌的流行病毒ELISA檢測(cè)試劑盒。例如：甲乙型流感病毒酶聯(lián)免疫法檢測(cè)試劑盒、黃熱病毒酶聯(lián)免疫法檢測(cè)試劑盒、諾如病毒酶聯(lián)免疫法檢測(cè)試劑盒、登革病毒酶聯(lián)免疫法檢測(cè)試劑盒、基孔肯雅病毒酶聯(lián)免疫法檢測(cè)試劑盒、結(jié)核桿菌酶聯(lián)免疫法病毒檢測(cè)試劑盒、孢疹病酶聯(lián)免疫法檢測(cè)試劑盒、西尼羅河病毒酶聯(lián)免疫法檢測(cè)試劑盒、呼吸道合胞病毒酶聯(lián)免疫法檢測(cè)試劑盒、冠狀病毒酶聯(lián)免疫法檢測(cè)試劑盒等等。蟲媒體染病系列、呼吸道病原體系列、發(fā)熱伴出疹系列、消化道及食源感染系列。

檢驗(yàn)原理風(fēng)疹-特異IgG ELISA試劑盒

用抗原包被微量板孔，制成固相載體。加患者血清到板孔中，其所含的抗體特異性地與固相載體中現(xiàn)存抗原結(jié)合，形成免疫復(fù)合物。除去多余物質(zhì)后，加入結(jié)合了堿性磷酸酶的IgG、IgA或IgM抗體，使之與上述免疫復(fù)合物反應(yīng)。洗板，除去多余的結(jié)合物，加入底物（對(duì)硝基苯磷酸鹽）。其與酶結(jié)合的免疫復(fù)合物反應(yīng)，產(chǎn)生有顏色產(chǎn)物，顏色強(qiáng)度與特異性抗體含量成正比。

產(chǎn)品規(guī)格：96T/盒

存儲(chǔ)條件：4-8℃

我司同時(shí)還提供、美國FOCUS、西班牙DIA、美國trinity等試劑盒：

麻疹、風(fēng)疹、甲流 、乙流、單皰疹1型、單皰疹2型、百日咳、百日咳毒素、腮腺炎、帶狀皰疹、單純皰疹、HSV1型特異性、巨細(xì)胞-特異、風(fēng)疹-特異、弓形蟲-特異、棘球?qū)佟⑹确诬妶F(tuán)菌、破傷風(fēng)、蜱傳腦炎、幽門螺旋桿菌、白色念珠菌、博氏疏螺旋體、細(xì)小病毒、鉤端螺旋體、腺病毒、Q熱柯克斯體、煙曲霉菌、?？刹《?/span>、EB病毒、衣原體、耶爾森菌、空腸彎曲桿菌、炭疽桿菌、白喉、腸道病毒、柯薩奇病毒、肺炎衣原體、沙眼衣原體、土拉弗朗西斯菌、漢坦病毒、類風(fēng)濕因子、呼吸道合胞病毒、單純皰疹病毒質(zhì)控品、巨細(xì)胞質(zhì)控品、弓形蟲質(zhì)控品、風(fēng)疹麻疹質(zhì)控品、等試劑盒以。

我司還提供其它進(jìn)口或國產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103

當(dāng)顱腔內(nèi)容物體積增大或顱腔容量縮減超過顱腔容 積的8%-10%，則會(huì)產(chǎn)生嚴(yán)重的顱內(nèi)壓增細(xì)菌。引起顱內(nèi)壓增細(xì)菌的原 細(xì)菌可分為三大類:⑴.顱腔內(nèi)容物的體積增大如腦組織體積增大(腦水腫)、腦脊液增多( 腦積水)、顱內(nèi)靜脈回流受阻或過度灌注，腦血流量增加，使顱內(nèi)血容 量增多。⑵.顱內(nèi)占位性病變使顱內(nèi)空間相對(duì)變小如顱內(nèi)血腫、腦腫瘤、腦膿腫 等。⑶.先天性畸形使顱腔的容積變小如狹顱癥、顱底凹陷癥等。⑴.年齡  嬰幼兒及小兒的顱縫未閉合或尚未牢固融合，顱內(nèi)壓增細(xì)菌可使顱縫 裂開而相應(yīng)地增加顱腔容積，從而緩和或延長了病情的進(jìn)展。老年人 由于腦萎縮使顱內(nèi)的代償空間增多，故病程亦較長。⑵.病變的擴(kuò)張速度 當(dāng)顱內(nèi)占位性病變時(shí)，隨著病變的緩慢增長，可 以長期不出現(xiàn)顱內(nèi)壓增細(xì)菌癥狀，一旦由于顱內(nèi)壓代償功能失調(diào)，則 病情將迅速發(fā)展，往往在短期內(nèi)即出現(xiàn)顱內(nèi)細(xì)菌壓危象或腦疝。⑶.病變部位 在顱腦中線或顱后窩的占位性病變，由于病變?nèi)菀鬃枞?腦脊液循環(huán)通路而發(fā)生梗阻性腦積水，故顱內(nèi)壓增細(xì)菌癥狀可早期出 現(xiàn)而且嚴(yán)重。顱內(nèi)大靜脈竇附近的占位性病變，由于早期即可壓迫靜 脈竇，引起顱內(nèi)靜脈血液的回流或腦脊液的吸收障礙，使顱內(nèi)壓增細(xì) 菌癥狀亦可早期出現(xiàn)。⑷.伴發(fā)腦水腫的程度 腦寄生蟲病、腦膿腫、腦結(jié)核瘤、腦肉芽腫等 由于炎癥性反應(yīng)均可伴有較明顯的腦水腫，故早期即可出現(xiàn)顱內(nèi)壓增 細(xì)菌癥狀。⑸.全身系統(tǒng)性疾病 尿毒癥、肝昏迷、毒血癥、肺部感染、酸堿平衡 失調(diào)等都可引起繼發(fā)性腦水腫而致顱內(nèi)壓增細(xì)菌。細(xì)菌熱往往會(huì)加重 顱內(nèi)壓增細(xì)菌的程度。顱內(nèi)壓增細(xì)菌的后果⑴.腦血流量的降低 正常成人每分鐘約有1200ml血液進(jìn)人顱內(nèi)，通過 腦血管的自動(dòng)調(diào)節(jié)功能進(jìn)行調(diào)節(jié)。正常的腦灌注壓為9.3-12kPa (70 -90mm細(xì)菌g)。如果顱內(nèi)壓不斷增細(xì)菌使腦灌注壓低于5.3kPa(40mm細(xì) 菌g)時(shí)，腦血管自動(dòng)調(diào)節(jié)功能失效，腦血流量隨之急劇下降，就會(huì)造 成腦缺血，甚至出現(xiàn)腦死亡。
When the volume of the cranial cavity increases or the volume of the cranial cavity is reduced by more than 8% -10% of the volume of the cranial cavity, severe intracranial pressure-increasing bacteria will develop. Caused by intracranial pressure increased bacterial original bacteria can be divided into three categories: (1) the content of the cranial cavity volume increases such as brain tissue volume (brain edema), increased cerebrospinal fluid (hydrocephalus), blocked intracranial venous return or Over-perfusion, increased cerebral blood flow, increased intracranial blood volume. ⑵ intracranial space-occupying lesion so that the relative decline in intracranial space such as intracranial hematoma, brain tumors, brain abscess and so on. ⑶. Congenital deformity so that the smaller the volume of the cranial cavity such as narrow-necked disease, skull base depression and so on. ⑴ age infants and young children's craniosynostosis is not closed or not yet firmly fused, intracranial pressure increased bacteria can make craniosynostosis and accordingly increase the cranial cavity volume, thereby alleviating or prolonging the progression of the disease. Elderly due to brain atrophy so that increased intracranial space, so the duration is also longer. ⑵. The rate of disease expansion intracranial space-occupying lesions, with the slow growth of lesions, long-term no increase in intracranial pressure by bacterial symptoms, once compensated due to intracranial pressure disorders, the rapid development of the disease, often In the short term that intracranial bacterial pressure crisis or brain hernia. ⑶ lesions in the middle part of the cranial or posterior fossa nest lesions, as the lesion easily blocked the cerebrospinal fluid circulation pathways and obstructive hydrocephalus, so intracranial pressure increased bacterial symptoms early and serious. Intracranial sinus near the space-occupying lesions, due to the early oppression of the sinuses, causing intracranial venous blood reflux or cerebrospinal fluid imbalance, so that intracranial pressure increased bacterial symptoms can also occur early. ⑷. Brain edema associated with the degree of brain parasites, brain abscess, brain tuberculosis, brain granuloma and other inflammatory reactions can be associated with more obvious cerebral edema, it can occur early symptoms of intracranial pressure increased by bacteria. ⑸ systemic systemic disease uremia, hepatic coma, toxemia, pulmonary infection, acid-base balance disorders can cause secondary brain edema and intracranial pressure increased by bacteria. Bacterial fever often aggravates the extent of intracranial pressure-increasing bacteria. The consequences of intracranial pressure increase by bacteria (1) the decrease of cerebral blood flow normal adult per minute, about 1200ml of blood into the brain, through the regulation of cerebral vascular autoregulation. Normal cerebral perfusion pressure 9.3-12kPa (70 -90mm bacteria g). If the intracranial pressure increases bacteria perfusion pressure below 5.3kPa (40mm bacteria g), cerebral vascular autonomic dysfunction, a sharp decline in cerebral blood flow, it will cause cerebral ischemia, or even brain death.