韓國SD瘧原蟲全血檢測試紙條

韓國SD瘧原蟲全血檢測試紙條

廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發(fā)、銷售、服務于一體的優(yōu)良企業(yè),公司產(chǎn)品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產(chǎn)品集團公司產(chǎn)品，致力于為商檢單位、疾病預防控制中心、海關出入境檢疫局、衛(wèi)生防疫單位,緝毒系統(tǒng)，戒毒中心，檢驗檢疫單位、生化企業(yè)、科研院所、醫(yī)療機構(gòu)等機構(gòu)與行業(yè)提供*、高品質(zhì)的產(chǎn)品服務。此外，本公司還開展食品、衛(wèi)生、環(huán)境、藥品等多方面的第三方檢測服務。）

韓國SD瘧原蟲全血檢測試紙條? 本試劑盒主要是采用膠體金層析的原理制成，用于檢測人體血清/血漿/全血標本中，感染的瘧原蟲抗體，包括了惡性瘧原蟲和間日瘧原蟲、卵形瘧原蟲、三日瘧原蟲共有抗原的鑒別性檢測。

人群易感性 人群對瘧疾普遍易感，感染后雖有一定的免疫力，但不持久，各型瘧疾之間亦無交叉免疫性，經(jīng)反復多次感染后，再感染時癥狀可較輕，甚至無癥狀，而一般非流行區(qū)來的外來人員常較易感染，且癥狀較重。
People susceptible to the crowd generally susceptible to malaria, although the infection after a certain degree of immunity, but not lasting, there is no cross-immunity between malaria, after repeated infections, re-infection symptoms may be lighter, or even Asymptomatic, while the non-endemic areas of non-migrant workers are often more susceptible to infection, and the symptoms are severe.

：

1 撕開檢測卡鋁箔袋，取出袋內(nèi)金標卡。注意：不要讓袋內(nèi)材料暴露于高溫高濕環(huán)境，撕開鋁箔袋后盡快使用。

2將金標卡平放在臺面上；并將病人名字和編號寫在標簽上。

3 取5微升(吸管*刻度處)全血標本，垂直加入金標卡上“加樣孔A”內(nèi)。

4 掰斷裂解液瓶子蓋子上方的綠色圓頭，在“樣品孔B”上垂直滴加4滴裂解液。

5 在十五分鐘內(nèi)出結(jié)果。注意：必須在15分鐘內(nèi)判讀結(jié)果，如超時判斷，結(jié)果無效。

6 請遵循相關法規(guī)，妥善處理樣本及廢棄材料。

7 存儲條件：2-30℃；

8 保質(zhì)期：18個月；

【病原學檢測】

瘧疾檢測,用于檢測出虐疾的病原體——瘧原蟲，是明確診斷的zui直接證據(jù)。目前常用的層析法，具有操作簡單、靈敏度高和可鑒別蟲種等優(yōu)點，廣泛用于瘧疾的病原學診斷，是目前zui常用的方法之一。

我司為美國NOVABIOS公司在中國地區(qū)戰(zhàn)略合作伙伴，負責該公司產(chǎn)品的總經(jīng)銷及售后服務工作。還與各疾控中心，疾病防御中心有合作關系，例如中國疾病預防控制中心 、浙江省疾病預防控制中心  ，詳情可以我司工作人員。

（  MOB：楊永漢）  

我司還提供其它進口或國產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

廣州健侖生物長期供應各種違禁品檢測試紙、違禁品檢測卡、違禁品檢測試劑盒、藥篩試紙、藥篩試劑盒、嗎啡檢測試劑盒、巴比妥檢測試劑盒等。

想了解更多的產(chǎn)品及服務請掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

【】 
【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103

多見于老年人，常與季節(jié)有關，皮損為繼發(fā)性。
5.原發(fā)性皮膚淀粉樣變
皮損呈高粱至綠豆大棕褐色堅硬丘疹，有時皮疹沿皮紋呈念珠狀

排列，組織病理上淀粉樣蛋白沉積具有特征性改變。扁桃體炎可

分為急性扁桃體炎和慢性扁桃體炎?；技毙詡魅静。ㄈ缧杉t熱、

麻疹、流感、白喉等）后，可引起慢性扁桃體炎，鼻腔有鼻竇感

染也可伴發(fā)本病。病源菌以鏈球菌及葡萄球菌等zui常見。臨床表

現(xiàn)為經(jīng)常咽部不適，異物感，發(fā)干、癢，刺激性咳嗽，口臭等癥

狀。膽囊炎是較常見的疾病，發(fā)病率較高。根據(jù)其臨床表現(xiàn)和臨

床經(jīng)過，又可分為急性和慢性兩種類型，常與膽石癥合并存在。

右上腹劇痛或絞痛，多見于結(jié)石或寄生蟲嵌頓梗阻膽囊頸部所致

的急性膽囊炎，疼痛常突然發(fā)作，十分劇烈，或呈絞痛樣。膽囊

管非梗阻性急性膽囊炎時，右上腹疼痛一般不劇烈，多為持續(xù)性

脹痛，隨著膽囊炎癥的進展，疼痛亦可加重，疼痛呈放射性，zui

常見的放射部位是右肩部和右肩胛骨下角等處。膽囊內(nèi)結(jié)石突然

梗阻或嵌頓膽囊管導致急性膽囊炎，膽囊管扭轉(zhuǎn)、狹窄和膽道蛔

蟲或膽道腫瘤阻塞也可引起急性膽囊炎。此外，增齡老化過程中

，膽囊壁逐漸變得肥厚或萎縮，收縮功能減退，造成膽汁淤滯、

濃縮并形成膽酸鹽；膽總管末端及Oddi括約肌變得松弛，容易發(fā)

生逆行性感染；全身動脈粥樣硬化，血液粘滯度增加可加重膽囊

動脈缺血。膽囊管或膽囊頸梗阻后，膽囊內(nèi)淤滯的膽汁濃縮形成

膽酸鹽，后者刺激膽囊黏膜引起化學性膽囊炎（早期）；與此同

時膽汁潴留使膽囊內(nèi)壓力不斷增高，膨脹的膽囊首先影響膽囊壁

的靜脈和淋巴回流，膽囊出現(xiàn)充血水腫，當膽囊內(nèi)壓>5.39kPa（

55cmH2O）時，膽囊壁動脈血流阻斷，膽囊發(fā)生缺血性損傷，缺血

的膽囊容易繼發(fā)細菌感染，加重膽囊炎進程，zui終并發(fā)膽囊壞疽

或穿孔。若膽囊管梗阻而沒有膽囊壁的血液循環(huán)障礙和細菌感染

，則發(fā)展為膽囊積液。近年的研究表明，磷脂酶A可因膽汁淤滯或

結(jié)石嵌頓從損傷的膽囊黏膜上皮釋放，使膽汁中卵磷脂水解成溶

血卵磷脂，后者進而使黏膜上皮細胞的完整性發(fā)生變化引起急性

膽囊炎。急性結(jié)石性膽囊炎的臨床表現(xiàn)和急性無結(jié)石性膽囊炎基

本相同。More common in the elderly, often associated with the seasons, skin lesions secondary.
5. Primary cutaneous amyloidosis
Lesions were sorghum to mung bean big brown hard papules, sometimes rashes were rosary beads

Permutations, histopathologically, amyloid deposits have characteristic changes. Tonsillitis can be

Divided into acute tonsillitis and chronic tonsillitis. Acute infectious diseases (such as scarlet fever,

Measles, flu, diphtheria, etc.), can cause chronic tonsillitis, nasal sinus feel

Dye can also be associated with the disease. Pathogens to Streptococcus and Staphylococcus are the most common. Clinical Table

Now often throat discomfort, foreign body sensation, hair dry, itchy, irritating cough, bad breath embolism

shape. Cholecystitis is a more common disease, with a higher incidence. According to its clinical manifestations and immediate

Bed after, can be divided into two types of acute and chronic, often combined with cholelithiasis.

Right upper quadrant pain or colic, more common in stones or parasite obstruction caused by gallbladder neck

Acute cholecystitis, often sudden onset of pain, very intense, or showed cramps. gallbladder

Non-obstructive acute cholecystitis, right upper quadrant pain is not usually intense, mostly persistent

Pain, with the progress of cholecystitis, the pain can be aggravated, the pain was radioactive, most

Common parts of the radiation is the right shoulder and right shoulder blade angle and other places. Gallbladder stones suddenly

Obstruction or incarceration of the cystic duct leads to acute cholecystitis, cystic duct torsion, stenosis and biliary cirrhosis

Obstruction of worms or biliary tumors can also cause acute cholecystitis. In addition, aging process

, Gallbladder wall gradually become hypertrophy or atrophy, systolic dysfunction, resulting in cholestasis,

Concentration and the formation of cholate; common bile duct sphincter and Oddi become loose, easy to hair

Health retrograde infection; systemic atherosclerosis, increased blood viscosity can aggravate the gallbladder

Arterial ischemia. Gallbladder or gallbladder neck obstruction, the formation of gallbladder stasis bile concentration

Cholic acid salt, which stimulates the gallbladder mucosa caused by chemical cholecystitis (early); the same

When bile retention to the gallbladder pressure continues to increase, the first expansion of the gallbladder wall gallbladder

Of venous and lymphatic drainage, congestion and edema of the gallbladder, when the gallbladder pressure> 5.39kPa (

55cmH2O), the gallbladder wall artery blood flow occlusion, ischemic injury of the gallbladder, ischemia

Gallbladder easily secondary to bacterial infection, aggravating cholecystitis process, the ultimate complication of gallbladder gangrene

Or perforated. If the cystic duct obstruction without gallbladder wall blood circulation disorders and bacterial infections

, Then develop into gallbladder fluid. Recent studies have shown that phospholipase A may be due to cholestasis or

Stones incarcerated from the injury of the gallbladder mucosal epithelium release, the bile lecithin hydrolysis

Blood lecithin, which in turn causes changes in the integrity of mucosal epithelial cells caused by acute

cholecystitis. Acute calculous cholecystitis clinical manifestations and acute calculi-based

The same.